Elyse Bolterstein

Elyse Bolterstein

Biology

Associate Professor

Office:  Room BBH 352A
Phone:  (773) 442-5742
Email:  e-bolterstein@neiu.edu
Office Hours:  Please email e-bolterstein@neiu.edu to arrange to meet or speak with Dr. Bolterstein.

Expertise

Molecular genetics and toxicology

General Biology 1 (BIO 201) 
Essential Skills for Biologists (BIO 250)
Genetics (BIO 303)
Biology Senior Seminar (BIO 390)
Biological Literature (BIO 405) 
Current Topics in Genetics (BIO 427) 

Research Interests

My research interests include the role of DNA repair proteins in aging, cellular interactions during tumorigenesis, and physiological responses to environmental toxins. Keywords: Drosophila, DNA repair, aging, DNA2, Drosophila Activity Monitor

Education

Ph.D. in Molecular and Environmental Toxicology from the University of Wisconsin-Madison

B.S. in Resource Ecology and Management from the School of Natural Resources and Environment, University of Michigan

Selected Publications

Undergraduate students are underlined

Krieger, K.L., Mann, E.K.,  Lee, K.G, Bolterstein, E., Jebakumar, D., Ittmann, M.M., Dal Zotto, V.L., Shaban, M., Sreekumar, A., Gassman, N.R.. (2023) Spatial mapping of the DNA adducts in cancer. DNA Repair. 128:103529.


Mitchell, C., Becker, V., DeLoach, J., Nestore, E., Bolterstein, E., & Kohl, K. P. (2022). The Drosophila Mutagen-Sensitivity Gene mus109 Encodes DmDNA2. Genes, 13(2), 312.


Epiney, D. G., Salameh, C., Cassidy, D., Zhou, L. T., Kruithof, J., Milutinović, R., Andreani, T. S., Schirmer, A. E., & Bolterstein, E. (2021). Characterization of Stress Responses in a Drosophila Model of Werner Syndrome. Biomolecules, 11(12), 1868.


Cassidy, D., Epiney D.G., Salameh, C., Zhou, L.T., Salomon, R.N., Schirmer, A.E., McVey, M., and Bolterstein, E. (2019) Evidence for premature aging in a Drosophila model of Werner syndrome. Experimental Gerontology. 127, 110733.


Bolterstein, E., Rivero, R., Marquez, M. and McVey, M. (2014) The Drosophila Werner exonuclease participates in an exonuclease-independent response to replication stress. Genetics. 197(2): 643-52.